#poison ivy Tumblr posts

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    Maybe you should just shut the fuck up.

    #dc #dc harley quinn series #dc poison ivy #poison ivy #dc harley quinn #harley quinn
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    This is some good high quality top tier gay content right here

    #harlivy #harley and ivy #dc harley quinn show #dc harley quinn #harley quinn #harley x ivy #poison ivy
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  • Love how only one of Batman’s villains has superpowers

    Everyone else was just someone with a really bad attitude and outlook on life, saw this dude dressing up as a bad to fight crime, and thought “I’m going to be crime” then picked an insane aesthetic

    #batman#poison ivy#dc comics #i dont count bane because hes taking super drugs to be strong #that doesnt count
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  • An open office document… A blinking cursor…

    So… I need to get back into writing and since Batman Rogues are currently a SpIn… I thought why not try to write some more content? I’m open to Headcanons and short drabbles/imagines. I only know a few rogues that I’m comfortable with writing things about however, it’s a very niche thing. The list goes as follows:

    Color codes : Green = Most Comfortable writing | Orange = Can write moderately well | Blue = Not much experience writing them but I have briefly in the past

    • Edward Nygma/Riddler : AU ; Gotham ; General
    • Jonathan Crane : AU ; Gotham ; Batman Begins ; General
    • Oswald Cobblepot : AU ; Gotham
    • Selina Kyle : AU ; General
    • Harley Quinn ; AU ; General ; SS/BoP
    • Poison Ivy ; General

    AU would either be my own AU or something stand alone.

    General is just the general character with no version specifically in mind.

    I typically write all the characters personalities as abstract with no specific version in mind. It only is needed when discussing appearances.

    I will also do character pairing posts!

    I know this is a short list, I’m sorry! Again, these are the ones I’m most comfortable with writing. You can 100% request me to write a character if you want me to write something about them, though I’ll politely request you don’t ask for any dealing with Joker.

    What should be included in your request:

    A general guideline

    1. What character
    2. Headcanon or Drabble
    3. If Headcanon: type of Headcanon/senario
    4. If Drabble: reader insert or character pairing?
    5. If reader insert: pronouns + gender
    6. If character pairing: what characters
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  • Batman and Harley Quinn (2017) #2

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  • Poison Ivy is a Sylph of Space.

    For determining the Classpects I looked at rosespecting’s Classpect Introspection guide, and Sylph Class Rundown, because while the Extended Zodiac does give info on Aspects, it doesn’t on Classes, and ultimately one wants to look at both (hence why it’s called a Classpect), and how the Class affects how one uses their Aspect. I’ll provide the descriptions given in the Guide and Rundown. Credit goes to rosespecting for these.


    Sylph :

    “Sylphs change their aspect. Unlike all other class pairs, the Witch and Sylph don’t really have too much or too little of their aspect. The Sylph has a strong desire to heal others with their aspect, but their low self-esteem can get in the way of that. Confident Sylphs may have issues accepting that someone wouldn’t benefit from their specific brand of help, and will try anyways.”


    Space :

    “Space is about the beginning. It’s enjoying every moment for what it is, not wanting to reach your destination if the path there is unpleasant. It’s art, creation, and birth. It’s burning down that which no longer has it’s place and making something better.”


    Sylph of Space :

    “The Sylph of Space does not often create something out of nothing like so many other Space players do, instead taking in full force towards the combination of various preexisting elements to make something new. When people are trying to hold onto what doesn’t work, they are there to remind them that not all things need to be fixed. Sometimes it is best to abandon what has not been working, and sometimes if you still wish to keep some part of it, the nature of it needs to be shifted entirely.”


    Canonically, the character that has this claspect is Kanaya Maryam. God Tier outfit for Sylph of Space:

    #enigmatic fear oc #enigmatic fear just nerd things #homestuck classpect#homestuck class#homestuck aspect #homestuck aspect space #homestuck class sylph #sylph of space #poison ivy#pamela isley #batman's rouges gallery
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  • Just Nerd Things #14

    Poison Ivy:

    #enigmatic fear oc #enigmatic fear just nerd things #pamela isley#poison ivy#dc comics #dc poison ivy #dc pamela isley #dr. pamela isley #homestuck#homestuck claspect #the extended zodiac #extended zodiac #extended zodiac virga #virga
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  • Going through questions:

    Nitroblue tetrazolium testing is used for diagnosis of Chronic Granulomatous Disease (CGD). Normally, neutrophils will turn blue on nitroblue tetrazolium testing because of the respiratory burst in phagolysosomes, but there is no blue color change in CGD because there is no production of oxidative species. CGD is due to an X-linked Recessive NADPH oxidase mutation. NADPH oxidase catalyzes production of reactive oxygen species, which kill bacteria. NADPH also catalyzes activation of granule proteases, like elastase, in phagosomes. Mutation of NADPH oxidase gene-> deficient NADPH oxidase-> neutrophils and macrophages can’t kill bacteria that they phagocytose. This leaves pts susceptible to recurrent fungal and bacterial infections, especially catalase + bugs (staph aureus, serratia marcenscens, aspergillus, burkholderia cepacia, and nocardia). The skin, lungs, liver, and lymph nodes are often infected in these pts. For diagnosis of CGD, you want to measure neutrophil superoxide production with either the Nitroblue Tetrazolium (NBT) test or with flow cytometry using dihydrorhodamine (DHR). The DHR test is better.

    IL-12 presented to naive helper T cells by macrophages causes naive helper T cells to differentiate into Th1 cells, which release IFN-gamma; IFN-gamma activates cytotoxic CD8+ T cells and macrophages to kill intracellular pathogens. Mycobacterium is an intracellular pathogen that is killed this way. People with IL-12 deficiency can’t mount a cell-mediated immune response and are susceptible to mycobacterium infection. Interferon gamma is given to treat these pts.

    In OnlineMedEd, Dustyn said that in graft vs host disease, the lymphocytes in the donor tissue trigger the recipient to make antibodies against the recipient. In this question I just answered, it says that T lymphs in the donated organ are sensitized to the recipient’s MHC Ag and then they attack the host’s tissues. Donor T cells from the donated organ go into the host’s tissues, become sensitized to the host’s MHC antigens, and then the donor CD4+ and CD8+ T cells destroy host cells. The GI tract, the skin, and the liver are usually affected. The pt can get a rash that includes the palms and soles.

    IL-2 is made by CD4+ helper T cells and activates more CD4+ helper T cells, cytotoxic CD8+ T cells, Naturak Killer cells, monocytes, and B cells. T cells and natural killer cells activated by IL-2 kill renal cancer and metastatic melanoma. I posted a picture showing how immumosuppressants work. So IL-2 causes activation of macrophages and NK cells, proliferation and differentiation of helper T cells, growth and secretion of IFN-gamma from T cells, and proliferation of B cells.

    Macrophages, B cells, dentritic cells = Antigen-Presenting Cells (APCs); they have MHC Class II, which they use to present angtigens to CD4+ helper T cells.

    Type IV (T cell-mediated/delayed type) HSR = T lymphs release cytokines that cause induration 24-48 hours after exposure to the Ag. So cytokines, CD8+ T cells, and macrophages cause the type IV HSR. Poison ivy is a type IV HSR.

    Poison ivy, poison oak, and poison sumac make urushiol, which attaches to proteins (haptenization)-> T-cell mediated immune response. CD8+ T cells destroy keratinocytes with the haptenated proteins.

    Accumulation of ADA-> destruction of lymphocytes. Adenosine is converted to inosine by adenosine deaminase (ADA). ADA also catalyzes the conversion of deoxyadenosine to deoxyinosine. If ADA is absent, then deoxyadenosine is converted to dATP and that causes lymphocyte apoptosis. Hairy cell leukemia is a lymphocyte cancer, which can be treated with ADA inhibitor (cladribine). ADA deficiency also causes SCID, which is lack of B and T cells.

    Hib vaccine is made with the polysaccharide capsule of Hib conjugated to tetanus toxoid carrier protein (or N. meningitidis outer membrane protein). The protein conjugation elicits T cell mediated immune response-> B cell activation-> memory B cells, which causes long term immunity. The capsule isn’t effective at eliciting the T cell response in pts younger than 2 years old because their humoral immunity is underdeveloped. So conjugated the capsule polysaccharide to a protein increases the humoral response against the capsule via T cell activation. T cells-> B cell stimulation-> memory B cells.

    Steroids cause neutrophil count to increase because it prevents them from marginating (lining up against the walls of blood vessels). Steroids can cause hypomania and psychosis. Steroids decrease basophils, eosinophils, lymphs, and monocytes.

    Mycophenolate inhibits IMPDH, which prevents conversion of inosine monophosphate to guanosine monophosphate. This prevents DNA and RNA synthesis in lymphs. Mycophenolate is used to prevent transplant rejection.

    Eosinophils release major basic protein, which causes the induration that occurs 2 to 10 hours after exposure to an allergen. It’s responsible for the late phase of a dermatologic type I HSR and is IgE-mediated. Major basic protein damages tissue. Wheal and flare is the early phase of a type I HSR in the skin. The late phase induration of the skin is due to release of major basic protein from eosinophils. Contrast this with type IV HSR, which is T cell-mediated and takes days, rather than hours, to develop.

    Classical complement pathway starts with C1 binding to 2 IgGs or 2 IgMs. C1 binds Fc region of heavy chain near the hinge point. After IgM binds to an antigen, the C1 binding site is revealed and C1 can bind to it, activating complement.

    There are more steps in WBCs leaving the vessels to get to the cytokines than what he mentioned in OnlineMedEd. The steps of inflammation are: margination, rolling, activation, tight adhesion and crawling, and transmigration. He grouped it differently in the video. Rolling is when the neutrophil slows down via interaction of its sialylated carbohydrate groups with selectins on endothelial cells. For example, sialyl Lewis X or PSGL-1 on neutrophils with selectins on endothelial cells. Then integrins on the endothelial surface (ICAM-1) stop the WBCs. In the tight adhesion and crawling phase of inflammation, CD18 beta 3 integrins on neutrophils bind to ICAM on endothelial cells. Then PECAM lets neutrophils slide through the spaces between the blood vessel wall cells during transmigration. Leukocyte adhesion deficiencies (LADs) prevent leukocytes from migrating from the blood to the site of cytokine release. LAD type 1 = no CD18-> no beta 2 intergins-> skin infections with no pus and delated separation of umbilical cord.

    C1 inhibitor deficiency leads to cleavage of C2 and C4-> excessive activation of complement system; also causes angioedema and GI symptoms.

    Hemolytic Disease of the Fetus and Newborn (HDFN) occurs when the mom has type O blood and thus makes IgG against type A and B blood. If this mom’s baby has type A, B, or AB blood, her IgG can diffuse across the placenta and cause hemolysis in the baby. But moms with type A or B blood make IgM antibodies (anti-B if the mom has type A blood; anti-A if the mom has type B blood), which can’t cross the placenta, and thus don’t cause hemolysis in the newborn. In contrast to Rh disease, HDFN can occur during the first pregnancy because the mom makes anti-B and anti-A IgGs in response to similar antigens that are encountered early in life.

    Leukotriene B4 causes neutrophils chemotaxis.

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  • My Eddie also has a huge crush on Ivy.  Ivy definitely knows it, and she humors him from time to time.  After all, he’s pretty cute.

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  • Harley Quinn and Poison Ivy: Summer Vibes

    Image from
    Harley Quinn #2 (2014)
    Written by Amanda Conner and Jimmy Palmiotti
    Art by Chad Hardin
    Colored by Alex Sinclair
    Lettered by John J. Hill
    Harley Quinn #8 (2016)
    Written by Amanda Conner and Jimmy Palmiotti
    Cover by Amanda Conner and Andrew Robinson
    Art by Chad Hardin and Bret Blevins
    Colored by Andrew Robinson
    Lettered by Dave Sharpe
    Harley Quinn & the Birds of Prey #1(2020)
    Written by Amanda Conner and Jimmy Palmiotti
    Art by Amanda Conner
    Colored by Paul Mounts
    Lettered by John J. Hill

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    Forgot to post Poison Ivy/Harley Quinn here

    #wlw#femslash#harley quinn#poison ivy#harleen quinzel#pamela isley#dc comics #lilies of ligbi #my art #comics mean blending iterations together because which outfit
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  • quick study with some fun colors

    #poison ivy#pamela isley #pamela lillian isley #poison ivy dc #dc poison ivy #batman poison ivy #tassie draws#woot woot#digital art#krita
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  • Almost done writing chapter 8/10, so good news is an update should be up later this week! This fic certainly won’t be complete by Sept. 22nd like I thought, but we’re getting close anyway. Besides, we’re deep in the spooky plot, so kind of on brand right now!

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  • Below imma put my reasoning for each of these if you were confused about why I put certain characters where I did.

    Keep reading

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  • poison ivy

    #rihanna#poison ivy #this is for someone #hope they see it
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